Just cutting off oxygen supply to the heart may not be enough to destroy the heart. The heart is one of the most innovative and resilient organs in the body, and it requires a lot of abuse for it to die. When the basement or basal membranes of the capillaries and arteries can no longer guarantee sufficient supply of oxygen, sugar, and insulin to the cells of the heart muscles, their ability to contract and pump blood is greatly reduced. Just to continue their work without enough oxygen, the heart cells begin to ferment glucose to produce energy, but this (anaerobic) process produces lactic acid, which acidifies the muscle tissues.

To continue maintaining its pumping action, the heart employs an additional emergency measure to gain energy, which is to mobilize and break down fats. Yet, without using oxygen in the process, these fats turn into harmful, cell-destructive acids. Proteins also begin to be used to provide energy; the by-products are once again harmful fatty acids. Since the thickening of the connective tissues as well as the lymph and blood capillaries in the heart begins obstructing normal elimination of metabolic waste, the heart muscles become intensely saturated with harmful acidic material. This may cause intense pain in the heart.

If uric acid, a waste product resulting from the breaking down of old cells, is retained in the tissues, gout occurs. The congestion leads to severe dehydration in the muscle cells, which prompts a group of cells known as mast cells to secrete the hormone histamine – a major water-regulating hormone in the body). When histamine passes over the sensitive pain nerves in the tissues, strong muscle pain results. If this form of muscle rheumatism occurs in the heart, it is called Angina pectoris. Both the acid accumulation and lack of oxygen lead to the death of the heart cells.

Heart attacks can occur in a number of ways:

1. The connective tissues surrounding the heart cells may become so densely congested that the heart cells simply die a painless death of suffocation.
2. In the case of an angina attack, it is acidification and low oxygenation of the heart muscles that destroys the heart.
3. The basal membranes of the capillaries and arteries are blocked and can no longer supply oxygen to the heart. The part of the heart where the attack occurs is also the part where the storage capacity for protein was first exceeded.
4. A blood clot breaks loose from a congested and injured blood vessel, enters the heart and blocks its oxygen supply.